[{"data":1,"prerenderedAt":-1},["ShallowReactive",2],{"$f65-g5LTUgHIk8dGZIHhGe2pKpxnfW0g-O4HGxk3yxcs":3,"$fPspxPVii4O1Rm6OhrYhnuwpTOOUYBKkYTC5dXQ0GG6w":8},{"code":4,"msg":5,"message":6,"data":6,"success":7},401,"认证失败，无法访问系统资源",null,false,{"code":9,"msg":10,"message":6,"data":11,"success":40},200,"操作成功",{"id":12,"name":13,"describes":6,"symptoms":6,"checkup":6,"treatment":6,"seoTitle":14,"seoKeywords":15,"seoDescription":16,"coverVertical":6,"coverAcross":6,"introduceClassifications":17,"departments":38},6161,"先天性视网膜劈裂症"," 先天性视网膜劈裂症的症状、诊断与治疗"," 先天性视网膜劈裂症, 视网膜疾病, 眼科医疗, 视网膜劈裂症治疗, 先天性眼病"," 了解先天性视网膜劈裂症的症状、诊断方法和治疗方案。详尽的医疗信息帮助患者及其家人更好地应对这种视网膜疾病。及时的诊疗对于保护视力至关重要。",[18,23,28,33],{"id":19,"classification":20,"description":21,"sort":22,"diseaseId":12},39696,"概述","\u003Cp>　　先天性视网膜劈裂症(congenital retinoschisis)属玻璃体-视网膜营养不良(vitreoretinal dystrophy)的一种，出生时就已存在，较获得性视网膜劈裂为少见，玻璃体积血和视网膜脱离是最严重的并发症。\u003C/p>",0,{"id":24,"classification":25,"description":26,"sort":27,"diseaseId":12},39700,"病因","\u003Cp>　　(一)发病原因\u003C/p>\u003Cp>　　本病是X性染色体隐性遗传病，但也有报道常染色体隐性遗传，常染色体显性遗传及遗传方式不确定。母亲为携带者，劈裂部位对称。\u003C/p>\u003Cp>　　(二)发病机制\u003C/p>\u003Cp>　　本病发病机制仍不确知，有玻璃体异常学说、Müller细胞缺陷学说及视网膜血管异常学说。由于视网膜最内层先天异常，特别是附着于内界膜的Müller细胞内端存在某种遗传性缺陷，或为玻璃体皮质异常，视网膜受其牵引，导致神经纤维层的分裂。这种牵引可能由于在正常发育眼球中玻璃体生长的不足，或在围生期玻璃体增厚与收缩。在胚胎期部分原发玻璃体与眼杯的内壁粘连，当原始玻璃体收缩时视网膜内层被牵引。视网膜颞侧下部分在胚胎晚期才开始发育，并且颞侧周边的血管发育晚，分布也较少。一旦视网膜内层受到牵引，则易在颞侧周边出现劈裂。\u003C/p>",1,{"id":29,"classification":30,"description":31,"sort":32,"diseaseId":12},39703,"症状","\u003Cp>　　常见症状：眼球震颤、中心暗点扩大、视力障碍\u003C/p>",2,{"id":34,"classification":35,"description":36,"sort":37,"diseaseId":12},39706,"检查","\u003Cp>　　检查项目：组织病理学检查、荧光血管造影\u003C/p>",3,[39],"眼科",true]